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Hire Dr. Rajesh V.

India

Seasoned Molecular Cancer Biologist, experience in DNA damage, tumor models of cancer, aging, genome instability,

Profile Summary

Trained Cancer biologist aim to deliver leading advancements in healthcare sector focusing on cancer biology, disease mechanism, drug discovery and molecular basis of drug resistance. I am particularly interested in molecular biology of cancer via dissect underlying molecular mechanism that leads to tumor onset, maintenance and development. I have experience of working with mouse and zebrafish model of cancer & Aging and genomic instability.

Subject Matter Expertise

• ★ Cell Aging
• ★ Drug Discovery
• ★ Cancer Research
• ☆ Molecular Biology
• ☆ Biomarkers
• ☆ Genetics
• ☆ Cell Biology
• ☆ Cell Signaling Pathways
• ☆ Skin Cancer

Services

• Writing
• Research
• Consulting
• Data & AI
• Product Development

Writing Clinical Trial Documentation, Technical Writing, Copywriting, Newswriting

Research Market Research, User Research, Meta-Research, Feasibility Study, Fact Checking, Gap Analysis, Gray Literature Search, Systematic Literature Review, Secondary Data Collection

Consulting Scientific and Technical Consulting

Data & AI Statistical Analysis, Data Visualization

Product Development Formulation, Stability/Shelf Life Testing, Product Evaluation, Material Sourcing, Product Validation, Manufacturing, Concept Development, Prototyping

Work Experience

Researcher

University of Minnesota

June 2018 - Present

Postdoctoral research associate

Umass Medical school

January 2014 - Present

Post doctoral research associate

Umass medical school

January 2014 - August 2016

Research Fellow

National Institute of Imunology

June 2006 - July 2008

Education

Ph.D.

VIB, K.U. Leuven, Belgium

January 2009 - June 2013

Ph.D. (Biomedical)

K.U. Leuven

January 2009 - June 2013

Certifications

• Certification details not provided.

Publications

JOURNAL ARTICLE

Potent inhibition of influenza virus replication with novel siRNA-chimeric-ribozyme constructs. Journal of general virology.

p53 attenuates AKT signaling by modulating membrane phospholipid composition. Onco target.

RNF4 is required for DNA double-strand break repair in vivo. Cell death and differentiation.

Cop1 constitutively regulates c-Jun protein stability and functions as a tumor suppressor in mice. Journal of clinical investigation.

Ligand-activated BMP signaling inhibits cell differentiation and death to promote melanoma. Journal of clinical investigation.

Nucleic acid-mediated cleavage of M1 gene of influenza A virus is significantly augmented by antisense molecules targeted to hybridize close to the cleavage site. Molecular biotechnology.

Reciprocal repression between P53 and TCTP. Nature Medicine.